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題名:阻力訓練造成肌肉生長效應:mTOR所扮演的角色
書刊名:大專體育
作者:許東雄 引用關係廖焜福 引用關係周麗卿陳宗與 引用關係
出版日期:2009
卷期:103
頁次:頁132-139
主題關鍵詞:運動訓練骨骼肌肌肉肥大蛋白質合成
原始連結:連回原系統網址new window
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  • 點閱點閱:103
期刊論文
1.Dreyer, H. C.、Fujita, S.、Cadenas, J. G.、Chinkes, D. L.、Volpi, E.、Rasmussen, B. B.(2006)。Resistance exercise increases AMPK activity and reduces 4E-BP1 phosphorylation and protein synthesis in human skeletal muscle。Journal of Applied Physiology,576(2),613-624。  new window
2.Baar, K.、Esser, K.(1999)。Phosphorylation of p70 (S6k) correlates with increased skeletal muscle mass following resistance exercise。American Journal of Physiology,276,120-127。  new window
3.Bodine, S. C.(2006)。mTOR signaling and the molecular adaptation to resistance exercise。Medicine and Science in Sports and Exercise,38(11),950-1957。  new window
4.Bodine, S. C.、Stitt, T. N.、Gonzalez, M.,、Kline, W. O.、Stover, G. L.、Bauerlein, R.、Yancopoulos, G. D.(2001)。Akt/mTOR pathway is a crucial regulator of skeletal muscle hypertrophy and can prevent muscle atrophy in vivo。Nature Cell Biology,3,1014-1019。  new window
5.Bolster, D. R.、Kubica, N.、Crozier, S. J.、Williamson, D. L.、Farrell, P. A.、Kimball, S. R.、Jefferson, L. S.(2003)。Immediate response of mammalian target of rapamycin (mTOR)-mediated signaling following acute resistance exercise in rat skeletal muscle。Journal of Physiology,553,213-220。  new window
6.Browne, G. J.、Finn, S. G.、Proud, C. G.(2004)。Stimulation of the AMPactivated protein kinase leads to activation of eukaryotic elongation factor 2 kinase and to its phosphorylation at a novel site, serine 398。Journal of Biological Chemistry,279,12220-12231。  new window
7.Cafferkey, R.、McLaughlin, M. M.、Young, P. R.、Johnson, R. K.、Livi, G. P.(1994)。Yeast TOR (DRR) proteins: Amino-acid sequence alignment and identification of structural motifs。Gene,141,133-136。  new window
8.Cross, D. A.、Alessi, D. R.、Cohen, P.、Andjelkovich, M.、Hemmings, B. A.(1995)。Inhibition of glycogen synthase kinase-3 by insulin mediated by protein kinase B。Nature,378(6559),785-789。  new window
9.Deldicque, L.、Theisen, D.、Francaux, M.(2005)。Regulation of mTOR by amino acids and resistance exercise in skeletal muscle。European Journal of Applied Physiology,94,1-10。  new window
10.Dennis, P. B.、Fumagalli, S.、Thomas, G.(1999)。Target of rapamycin (TOR): Balancing the opposing forces of protein synthesis and degradation。Current Opinion in Genetics and Development,9,49-54。  new window
11.Farrell, P. A.、Fedele, M. J.、Vary, T. C.、Kimball, S. R.、Lang, C. H.、Jefferson, L. S.(1999)。Regulation of protein synthesis after acute resistance exercise in diabetic rats。American Journal of Physiology,276,721-727。  new window
12.Hardie, D.、Salt, I.、Hawley, S.、Davies, S.(1999)。AMP-activated protein kinase: An ultrasensitive system for monitoring cellular energy charge。Journal of Biochemistry,338(Pt. 3),717-722。  new window
13.Huang, S.、Houghton, P. J.(2003)。Targeting mTOR signaling for cancer therapy。Current Opinion in Pharmacology,3,371-377。  new window
14.Inoki, K.、Zhu, T.、Guan, K. L.(2003)。TSC2 mediates cellular energy response to control cell growth and survival。Cell,115,577-590。  new window
15.Kimball, S. R.、Farrell, A.、Jefferson, L. S.(2002)。Invited review: Role of insulin in translational control of protein synthesis in skeletal muscle by amino acids or exercise。Journal of Applied Physiology,93,1168-1180。  new window
16.Li, Y.、Corradetti, M. N.、Inoki, K.、Guan, K. L.(2004)。TSC2: Filling the GAP in the mTOR signaling pathway.。Trends in Biochemical Sciences,29,32-38。  new window
17.Nader, G. A.、Mcloughlin, T. J.、Esser, K. A.(2005)。mTOR function in skeletal muscle hypertrophy: Increased ribosomal RNA via cell cycle regulators。American Journal of Physiology-Cell Physiology,289,1457-1465。  new window
18.Nave, B. T.、Ouwens, M.、Withers, D. J.、Alessi, D. R.、Shepherd, P. R.(1999)。Mammalian target of rapamycin is a direct target for protein kinase B: Identification of a convergence point for opposing effects of insulin and amino-acid deficiency on protein translation。Biochemical Journal,344(2),427-431。  new window
19.Proud, C. G.(2002)。Regulation of mammalian translation factors by nutrients。European Journal of Biochemistry,269,5338-5349。  new window
20.Raught, B.、Gingras, A. C.、Sonenberg, N.(2001)。The target of rapamycin (TOR) proteins。Proceedings of the National Academy of Sciences,98,7037-7044。  new window
21.Rennie, M. J.、Wackerhage, H.、Spangenburg, E. E.、Booth, F. W.(2004)。Control of the size of the human muscle mass。Annual Review of Physiology,66,799-828。  new window
22.Rutter, G.、Da Silva Xavier, G.、Leclerc, I.(2003)。Roles of 5'-AMP-activated protein kinase (AMPK) in mammalian glucose homoeostasis。Journal of Biochemistry,375(Pt 1),1-16。  new window
23.Sarbassov, D. D.、Ali, S. M.、Kim, D. H.、Guertin, D. A.、Latek, R. R.、Erdjument-Bromage, H.、Sabatini, D. M.(2004)。Rictor, a novel binding partner of mTOR, defines a rapamycininsensitive and raptor-independent pathway that regulates the cytoskeleton。Current Biology,14,1296-1302。  new window
24.Tee, A. R.、Manning, B. D.、Roux, P. P.、Cantley, L. C.、Blenis, J.(2003)。Tuberous sclerosis complex gene products, Tuberin and Hamartin, control mTOR signaling by acting as a GTPase-activating protein complex toward Rheb。Current Biology,13,1259-1268。  new window
25.Williamson, D. L.、Kubica, N.、Kimball, S. R.、Jefferson, L. S.(2006)。Exercise-induced alterations in extracellular signal-regulated kinase 1/2 and mammalian target of rapamycin (mTOR) signalling to regulatory mechanisms of mRNA translation in mouse muscle。Journal of Physiology,573,497-510。  new window
26.Yonezawa, K.(2003)。Identification of TOR-interacting proteins。Molecular Interventions,3,189-193。  new window
27.Nader, G. A.、Esser, K. A.(2001)。Intracellular signaling specificity in skeletal muscle in response to different modes of exercise。Journal of Applied physiology,90(5),1936-1942。  new window
 
 
 
 
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