目的：考驗「人體肘屈肌群（EF）做最大離心運動引起肌肉損傷，是否會引起肱動脈硬化及血流量增加」之假設。方法：讓15名健康大學男性學生的非慣用EF，在進行30次最大等速（30°/s）離心收縮運動前與後第0-4天，各接受一次：最大等長肌力、肌肉酸痛、上臂圍、肱動脈脈動指數、阻抗指數、心跳率與血流量的測驗。結果：在最大離心收縮後肌肉損傷恢復期間，最大等長肌力、肌肉酸痛、上臂圍、肱動脈脈動指數與血流量引起顯著改變，但到了最大離心收縮後第4天還未恢復至前測值（p<.05）；但在最大離心收縮前後之間，心跳率與阻抗指數無引起顯著變化。結論：這些結果顯示，在做最大離心收縮所產生的高張力會對肌肉和血管組織引起細微損傷，並在最大離心收縮隨後啟動發炎反應，進而藉由增加肱動脈血流量提供更多相關營氧物質，以利於損傷組織進行修復。

Purpose: To test the hypothesis that maximal eccentric exercise-induced muscle damage of the elbow flexors would produce significant changes in brachium artery stiffness and blood flow volume. Methods: Fifteen untrained young men performed a bout of 30 maximal isokinetic (30°/s) eccentric contractions of the elbow flexors of the non-dominant arm. Maximal isometric strength, muscle soreness, upper arm circumference, pulsatility index, resistance index, blood flow volume and heart rate measurements were taken before, immediately after, and for 4 consecutive days after maximal eccentric contractions. Results: Significant changes were noted in maximal isometric strength, upper arm circumference, muscle soreness, blood flow volume and pulsatility index following maximal eccentric exercise, and these variables had still not returned to baseline levels at 4 days after maximal eccentric contractions (p<.05). No significant changes in heart rate and resistance index following maximal eccentric contractions were observed compared with pre-maximal eccentric contractions levels. Conclusion: These results suggest that the high tension produced during maximal eccentric exercise to the exercised muscles (e.g. maximal isometric strength loss) and blood tissues (decrease in pulsatility index) would produce microtrauma, launching a series of inflammatory response events. This in turn would lead to further increases in artery blood flow volume, in order to gain more oxygen and nutrient materials for the repair/regeneration of damaged tissue.